Please use this identifier to cite or link to this item: https://repositorio.ufpa.br/jspui/handle/2011/10928
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dc.creatorSOUZA, Dilma do Socorro Moraes de-
dc.creatorARAUJO, Marialva Tereza Ferreira de-
dc.creatorSANTOS, Paulo Roberto Silva Garcez dos-
dc.creatorFURTADO JUNIOR, Julio Cesar Branco-
dc.creatorFIGUEIREDO, Maria Tereza Sanches-
dc.creatorPÓVOA, Rui Manuel dos Santos-
dc.date.accessioned2019-04-09T17:49:42Z-
dc.date.available2019-04-09T17:49:42Z-
dc.date.issued2016-07-
dc.identifier.citationSOUZA, Dilma do Socorro Moraes de et al. Anatomopathological aspects of acute Chagas myocarditis by oral transmission. Arquivos Brasileiros de Cardiologia, São Paulo, v. 107, n. 1, p. 77-80,  July 2016. DOI: http://dx.doi.org/10.5935/abc.20160110. Disponível em: http://repositorio.ufpa.br/jspui/handle/2011/10928. Acesso em:.pt_BR
dc.identifier.issn1678-4170pt_BR
dc.identifier.urihttp://repositorio.ufpa.br/jspui/handle/2011/10928-
dc.description.abstractVector transmission of Trypanosoma cruzi has declined in Latinpt_B America, which has been attributed to better epidemiological control of this form of transmission, estimated at 8 to 10 million chronic cases, in addition to reducing the number of new cases.1,2 However, there has been an increase in the incidence of acute cases, predominantly by oral transmission due to the ingestion of food contaminated with feces of triatomids, both in isolated cases and in family micro-outbreaks.3 Necropsy studies that describe myocarditis in the acute phase of Chagas' disease are scarce and the existing reports in the literature are of studies carried out in the past decades and involve vector transmission in endemic areas. Cardiac involvement in the acute phase may have varied aspects, especially in relation to myocardial lesion, from an undetectable one, to the evolution to acute heart failure with severe myocarditis and death. Cardiac involvement is present in 90% of the cases, manifesting with myocarditis and pericardial effusion.4,5 The analysis of a series of acute myocarditis cases showed a mortality of 5.6%.en
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dc.description.provenanceMade available in DSpace on 2019-04-09T17:49:42Z (GMT). No. of bitstreams: 3 license_rdf: 0 bytes, checksum: d41d8cd98f00b204e9800998ecf8427e (MD5) Article_AnatomopathologicalAspectsAcute.pdf: 527764 bytes, checksum: 422349039a78dcb4dee0d7765c976d0e (MD5) Artigo_AspectosAnatomopatologicosMiocardite.pdf: 492425 bytes, checksum: 4708306fd92fe9598c700a4e8f536eb9 (MD5) Previous issue date: 2016-07en
dc.languageporpt_BR
dc.languageengpt_BR
dc.publisherSociedade Brasileira de Cardiologiapt_BR
dc.relation.ispartofArquivos Brasileiros de Cardiologiapt_BR
dc.rightsAcesso Abertopt_BR
dc.source.urihttp://ref.scielo.org/4bqpdxpt_BR
dc.subjectChagas cardiomyopathyen
dc.subjectPathologyen
dc.subjectChagas diseaseen
dc.subjectTransmissionen
dc.subjectTrypanosoma cruzipt_BR
dc.subjectFood parasitologyen
dc.titleAnatomopathological aspects of acute Chagas myocarditis by oral transmissionen
dc.title.alternativeAspectos anatomopatológicos da miocardite chagásica aguda por transmissão oralpt_BR
dc.typeArtigo de Periódicopt_BR
dc.publisher.countryBrasilpt_BR
dc.publisher.initialsSBCpt_BR
dc.creator.Latteshttp://lattes.cnpq.br/7368351594360330pt_BR
dc.creator.Latteshttp://lattes.cnpq.br/6779304013585161pt_BR
dc.creator.Latteshttp://lattes.cnpq.br/8980880025239288pt_BR
dc.creator.Latteshttp://lattes.cnpq.br/0720260344186762pt_BR
dc.creator.Latteshttp://lattes.cnpq.br/0775554226874282pt_BR
dc.creator.Latteshttp://lattes.cnpq.br/2613638579389329pt_BR
dc.citation.volume107-
dc.citation.issue1-
dc.citation.spage77-
dcterms.citation.epage80-
dc.identifier.doihttp://dx.doi.org/10.5935/abc.20160110pt_BR
dc.description.resumoA transmissão vetorial do Tripanossoma cruzi tem diminuído em países da América Latina, o que tem sido atribuído ao melhor controle epidemiológico dessa forma de transmissão, estimada em 8 a 10 milhões de casos crônicos, além da redução do número de casos novos.1,2 No entanto, ocorre o crescimento da incidência de casos agudos, predominando a transmissão por via oral, devido à ingesta de alimentos contaminados com fezes de triatomíneos, tanto em apresentações de casos isolados quanto em surtos de microepidemia familiar.3 Estudos de necrópsia que descrevem a miocardite na fase aguda da doença de Chagas são escassos, e os relatos existentes na literatura são de décadas passadas e envolviam a transmissão vetorial nas áreas endêmicas. O comprometimento cardíaco na fase aguda pode ter aspectos variados, principalmente em relação à lesão miocárdica, desde inaparente até evolução para insuficiência cardíaca aguda, com miocardite grave e óbito. O envolvimento cardíaco está presente em 90%, manifestando-se com miocardite e derrame pericárdico.4,5 A análise de série de casos de miocardite aguda apresentou mortalidade de 5,6%.pt_BR
dc.description.affiliationARAUJO, M. T. F.; SANTOS, P. R. S. G.; FURTADO JUNIOR, J. C. B.; FIGUEIREDO, M. T. S. Universidade Federal do Parápt_BR
Appears in Collections:Artigos Científicos - FAMED/ICS

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